EFEK SAMPING HALOPERIDOL PDF

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diltiazem, enalapril, phenytoin, fluconazole, haloperidol, itraconazole, macrolides, nifedipine, quinidine, rapamycin, simvastatin, theophylline, and verapamil. Haloperidol adalah obat yang dikategorikan ke dalam agen antipsikotik, antidiskinetik, Efek samping haloperidol berbeda pada berbagai tingkatan usia . efek. Pada pasien skizofrenia penurunan konsentrasi haloperidol 70 % sehingga efek antipsikosis menurun. Litium dapat meningkatkan efek samping peningkatan.

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Severe toxic CNS depression; preexisting coma; Parkinson’s disease; lactation. Download full text Bahasa Indonesia, 6 pages. Those levels normalize as ssamping treatment continues. Mandell, Douglas, and Bennett’s principles and practice of infectious diseases.

In vitro resistance to ethambutol develops slowly and is probably due to mutations in the embB gene.

Perbandingan Risperidone Dengan Haloperidol

Ethambutol inhibits the arabinosyltransferase enzyme encoded by the embB gene, which mediates the polymerization of arabinose into arabinogalactan. These drugs are metabolized in the liver.

Think your supplement is safe? Risperidone dominant over haloperidol in schizophrenia Documents.

Haloperidol: Indication, Dosage, Side Effect, Precaution | Indonesia

Tuberculosis and diabetes mellitus: Tuberculosis; Drug interactions; Antibiotics, antitubercular; Pharmacologic actions; Drug toxicity; Drug-induced liver injury. Europe and United States: The concentration sampint during meningitis.

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In patients using isoniazid in isolation, the risk of developing hepatitis increases with age. May increase plasma levels of haloperidol when used with hakoperidol or chlorpromazine. The use of rifampin in isolation can induce cholestasis, since the drug is excreted in bile after having been absorbed by the hepatocyte and having undergone partial deacetylation. Use in patients with liver failure.

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Perbandingan Risperidone Dengan Haloperidol

Non experimental, retrospective and Case Control study design was done. These levels decrease spontaneously as the treatment progresses. Isoniazid drug and food interactions. The genetics and biochemistry of isoniazid resistance in mycobacterium tuberculosis. There is a decrease in the plasma concentrations of the following drugs when administered concomitantly with rifampin: Therefore, rifampin can increase the metabolism of numerous drugs that are partially or completely metabolized by CYP when these drugs are administered concomitantly with rifampin.

Oral Restlessness and confusion Adult: Ocular toxicity from ethambutol. If exanthema occurs, treatment should be discontinued, and the drugs should be subsequently reintroduced, one by one, in order to identify the causative drug. Maintain a patent airway by using an oropharyngeal airway or endotracheal tube or, in prolonged cases of coma, by tracheostomy. N Engl J Med. Relationship with rifampicin therapy in two cases [Article in French].

The possibility of interaction between rifampin and other drugs calls for a thorough history taking that focuses on the drugs currently used by patients. Use in patients with kidney failure. Gastrointestinal reactions can be treated as previously described for isoniazid. The serum half-life of ethambutol is h, and it can be as long as 10 h in patients with severe kidney failure. Isoniazid is a hepatotoxic drug, the effect of which becomes more evident in individuals with liver disease, in alcoholic individuals, and in individuals over 50 years of age.

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Rhabdomyolysis with myoglobinuria and kidney failure is a rare complication of pyrazinamide treatment and requires that the drug be discontinued.

The administration of rifampin in isolation rarely causes liver changes.

IM Acute psychosis mg hrly or hrly until symptoms are controlled. Therapeutic uses and prescribing information may vary between countries. Ethambutol interferes with the biosynthesis of arabinogalactan, the principal polysaccharide on the mycobacterial cell wall.

Arthralgia is a rare complication of isoniazid administration and responds to treatment with nonsteroidal antiinflammatory drugs. The most common mechanism of resistance to isoniazid consists of KatG mutations, which decrease the activity of isoniazid and prevent the prodrug from being converted into its active metabolite.